EMF Blocking in addition to EMF Shielding
No matter if you live in a house or apartment, or you just would like to ensure your home is free from EMFs There are plenty options to limit your exposure. One of the easiest is to restrict your use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from reaching your house. Another method to protect your house against EMF radiation is to put up a shielding canopy for RF. This is a fabric of net that contains EMF shielding. It's used to block EMFs from entering rooms. Another alternative is to have your home fitted with a conductive enclosure. These devices are called Faraday cages.
Several studies have shown how the non-ionizing energy of RF has antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to involve down-regulation the cancer stem cell. This could be the reason for the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on cancer patients is not evident.
Effects of AM RF EMF on HCC tumour growth in vivo were examined in mice. The tumours were divided into 3 groups. First, block emf that was unaffected RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to the one used by humans. In emf blocker , they were subjected the RF EMF in HCC-specific frequencies. The effects of HCCMF on tumours was evaluated against that of RCF. The results showed that the tumors treated by HCCMF were significantly shrinking. However, the tumours treated with RCF didn't show evidence of tumour shrinkage.
The mechanism behind cancer-specific AM RF EMF could be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is controlled by CACNA1H which is a protein which regulates the Ca2+ influx specific to tumors. The findings suggest that CACNA1H could have more broader implications for the treatment and diagnosis of many cancers.
The tumors in those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumors in those in the HCCMF group were infected with Huh7 cells when they were between five and seven weeks old. The tumors were then killed when they showed excessive burden.
The tumors of the three groups also displayed different growth curves. The HCCMF-treated tumors saw a significant reduction in tumour size after eight weeks. However, the tumors that were treated using RCF showed no signs of shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis, which is common when tumors are exposed to RCF. There is a possibility that the necrosis is caused by a lack of oxygen in the more invasive tumours.
In conclusion, the findings indicate that AM RF EMF exhibits anticancer effects in vitro and in the vivo. Learn more have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage for HCC patients. The possibility is that the AM EMF produces these effects due to CACNA1H which is a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the development of HCC tumors in the vivo.